1·Objective:To illustrate mechanisms of arsenic trioxide(As2O3) in the treatment of acute promyelocytic leukemia(APL).
目的：阐明氧化砷治疗急性早幼粒细胞白血病（APL）的机制。
2·Objective to investigate the effect of bortezomib alone and in combination with arsenic trioxide on apoptosis of HL-60 cells.
目的探讨硼替佐米单独或联合了氧化二砷对HL - 60细胞凋亡的影响。
3·Conclusion: Suitable arsenic trioxide concentration can damage nerve axons permanently, and can not bring side effect to the structure around the nerve and whole body.
结论：适当浓度的亚砷酸可引起神经轴突的损害，且对全身及周围软硬组织无毒性破坏作用。
4·Objective: to investigate the apoptosis inducing function of arsenic trioxide on CML cell line K562 in vitro and elucidate the possible cellular and molecular mechanisms.
目的：研究三氧化二砷体外对K562细胞凋亡的诱导作用，探讨三氧化二砷促进慢粒细胞系k562细胞凋亡的机制。
5·The production of arsenic trioxide is controlled by electrics and instrument, with all electrical equipment, units and valves automatically controlled by DCS or remote operation.
亚砷酸车间的生产控制采用仪电一体化设计，所有电气设备、机组和阀门均在DCS上自动控制或远方遥控操作。