1·This is mainly due to the inhibition of glycosylation of newly synthesized insulin receptor by tunicamycin.
这主要由于衣霉素抑制新合成的胰岛素受体的糖基化所致。
2·Conclusion: Tunicamycin could induce RPMI-8226 cell differentiation for mature stage.
结论:衣霉素可使RPMI - 8226细胞向成熟阶段分化。