1·Just why active cytomegalovirus is associated with glioblastoma is still unclear.
活跃的巨细胞病毒和胶质母细胞瘤相联系的原因还不清楚。
2·Objective: to investigate the variance and significance of expression of P53 and PCNA proteins in recurrent glioblastoma after gamma knife treatment.
目的：初步探讨伽玛刀治疗前后复发胶质母细胞瘤中P 53及PC NA蛋白表达的变化及其临床意义。
3·C6 cell line and human glioblastoma cells contained 1% cells which were ABCG2 positive.
C6细胞系和人胶质母细胞瘤细胞中均发现有1%左右的细胞ABCG2蛋白表达阳性。
4·This may supply the new target point and strategy for the apoptosis-induced therapy of glioblastoma.
这可能为胶质母细胞瘤的凋亡诱导治疗提供新的靶点和新的策略。
5·Conclusion RCAS1 is a new tumor-associated antigen of human astrocyte tumors, whose expression correlated to the malignant transformation of glioblastoma.
结论R CAS1是人脑星形细胞瘤的新的肿瘤相关抗原，其表达与胶质母细胞瘤的形成相关。
1·Most standard clinical treatments for glioblastoma currently target just one pathway.
而大多数现有的针对恶性胶质瘤的临床治疗方式只作用于一条通路。
2·Eberhart and colleagues studied glioblastoma cell lines to investigate the effects of a gamma-secretase inhibitor, a medication that blocks the Notch receptor, on tumor growth.
埃伯哈特和他的同事们研究了恶性胶质瘤的细胞系以发现γ-内分泌酶抑制剂的影响，这种药物可阻断促使肿瘤增长的Notch受体途径。
3·Researchers discovered that switching off the RAD51 molecule increased the effectiveness of radiotherapy to kill glioblastoma cells in the lab.
研究人员发现，在实验室关闭RAD51分子，杀死恶性胶质瘤细胞，提高了放射治疗的效果。